I think separating out the primary and secondary agencies in the tricky bit, Dave. When a tree's in a bad enough way for it to fail, I generally observe more than one agent present. When you have a syndrome of a number of potential pathogens in a tree, whose to say which is going to be the straw that broke the camel's back? I looked at a beech the other week (which I have to go back and PiCUS, which is showing early Kd, Meripilus (a rare local case) and a young Ganoderma. If it were to go over, could we be 100% sure which one did it? I don't know.
With respect to the spalting, I condemned a sycamore literally within falling distance of my humble office which was riddled with Kd and the spalting went all the way up the first break at about 8 metres. The stumps's still there, so I could post an image of it. I must admit, it was a very advanced case, with obvious fruiting bodies up above head height.
In terms of beech with full root plate failure as was shown in these images, I have certainly seen a good number, beech and sycamore (less so) around here where the key visible causal agent was Kd, with fruiting bodies being seen and the remaining roots being a mess of filmy sheets of black chitin. Like you suggest the Kd could be working either saprophytically, or maybe in a syndrome with other fungi for which direct evidence wasn't visible. It could be that they fail earlier in the decay process here because it's extremely windy. Who knows?
I've always found the literature on tree failure pretty patchy, vague, often contradictory and frequently prone to generalisation. One of the better sources I'd go to, David Lonsdale, reckons the following:
Extract from ‘Principles of tree hazard assessment and management’ by David Lonsdale 1999:
“….K.deusta induces a brittle ceramic-like fracture. This can occur in main stems or root systems, since the fungus is exceptional amongst ascomycetes in being able to grow in the central wood of very large trees.
Fracture often occurs before an advanced white-rot has developed, so that the fracture surface can be quite hard.
The seat of the decay within the tree is usually at the stem base, where in some cases the fungus appears to have entered through a wound. In such cases, it can extend 4m or more up the stem, as well as into the roots. It can also enter via the roots, eventually causing windthrow.
This is a particularly dangerous decay fungus, partly because its fruit bodies are often overlooked, also because of its very common occurrence and wide host range, and finally because of the type of decay that it causes. The brittle fracture associated with this decay often occurs with no warning of incipient failure, and without the compensatory thickening that can occur with fungi which cause selective delignification (e.g. Ganoderma spp.). Except in very advanced cases, this decay cannot be detected with a stress wave timer and may also escape detection by certain kinds of mechanical probe.”